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STAT1, a core regulator of interferon signaling, drives MAFLD-to-NASH progression ( Semaglutide elevated branched-chain amino acids (BCAAs: valine, leucine, isoleucine), potentially inhibiting lipogenic genes (e.g., SREBP-1c) via AMPK activation while enhancing -oxidation ( Integrated multi-omics analysis demonstrated semaglutides predominant effects on fatty acid metabolism and NAFLD-related pathways

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TGAb were present at a titer of 1/160 and TPOAb at 1/20480