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Interaction of TBC1D9B with mammalian ATG8 homologues regulates autophagic flux

Este cambio metablico se debe a la accin del pptido sobre la va de metilacin de la nicotinamida , que aumenta la disponibilidad intracelular de NAD+, facilitando as un mayor metabolismo oxidativo

This represents a common pattern in pharmaceutical development, where nature provides the initial insight that leads to entirely synthetic therapeutic solutions