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Hirschberg, A

Given the role of the caudal hindbrain in emetic/aversive behavior (191) and in mediating GLP-1 effects on food intake (192, 193), these data collectively suggest that GIP potentially acts on the hindbrain-hypothalamus axis to decrease food intake and to improve tolerability of GLP-1R agonism

We therefore set out to show the applicability of daLUXendin660 for TIRF-based GLP1R/GIPR single particle tracking at the cell membrane in live MIN6-CB4 cells (Fig

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