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discovered that the lactylation of NBS1 at lysine 388 (K388) can promote the accumulation of the MRN complex and HR proteins at the site of DNA double-strand breaks (DSBs), thereby facilitating DNA damage repair, which is frequently associated with poor outcomes of tumor patients undergoing neoadjuvant chemotherapy [27]

Based on your consultation findings, we recommend a specific treatment protocol

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Therefore, we conducted a 20-week study, testing five different levels of inclusion of muramidase, from 0 mg/kg to 600 mg/kg

Gaitan 2012)

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