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and Fahd Al-Mulla for their contribution to the peer review of this work

In the kidneys, activin is overexpressed after injury and during kidney fibrosis, mimicking TGF- signaling and hampering regeneration 103 , but follistatin blockade promotes epithelial proliferation and repair 104

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Cutting edge: distinct glycolytic and lipid oxidative metabolic programs are essential for effector and regulatory CD4+ T cell subsets