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Thus, targeting apoC3 or apoA5 seems to be a potential approach to reduce plasma RC levels, which could be testified in future trials

The etiology of IBD is multifactorial and results from a complex interplay between mucosal immunity, environmental factors, and host genetics

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Strict monitoring in clinical trials which enhance medication efficacy and patient compliance are generally not replicated in real-world settings

N-acetylcysteine protects against myocardial ischemia-reperfusion injury through anti-ferroptosis in type 1 diabetic mice

One of the pathways is a free radical-mediated Maillard reaction