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Helps with cognitive function and nerve stability

Notably, breast cancer cells achieve metabolic adaptation by hijacking the autophagy mechanism: under glucose-deprivation conditions, MANF-mediated mitophagy eliminates dysfunctional mitochondria through the PRKN/Parkin-dependent pathway, maintains the ATP supply and promotes tumour survival [112], suggesting that targeting the MANFPRKN interaction can break the drug resistance barrier of breast cancer under metabolic stress

Researchers believe that activating GLP-1 receptors may reduce inflammation, improve brain cell health, and help regulate pathways linked to stress and anxiety

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Every year, we will add capacity for small-molecule APIs and peptides, Chen says