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doi: 10.3390/toxins13080586, 27

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doi: 10.3390/toxins16010006 63 LiuJ.ZhaoW.HuC.XiaY.LiL.ZhangF.et al

For example, the p53 mutants p53 R 237 H and p53 R 175 H promote sensitivity to ferroptosis-like cell death, most likely through the combination of p53 mutants with NRF2 and through the suppression of the NRF2-dependent transactivation of SLC7A11 together with other antioxidant genes that oppose ferroptosis (Sasaki et al., 2002

The changes in SOD activity in a particular compartment may lead to the generation of a hydrogen peroxide concentration gradient and the subsequent activation of redox sensitive pathways

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