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Silencing MC5-R in cardiomyocytes renders mice susceptible to cardiac hypertrophy and fibrosis after pressure overload

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This data is significant because it is the first robust evidence we have seen that shows what happens to GLP-1 and GIP levels in non-diabetic patients with moderate to severe kidney disease after they have a meal, explained Armin Ahmadi, postdoctoral scholar of nephrology at UC Davis Health and lead author of the study