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[7] Oxidative stress and fatigue Increased levels of inflammatory cytokines can deplete levels of the key detoxification enzyme glutathione

MOTS-c improves metabolic flexibility, insulin sensitivity, and cellular energy regulation by acting on mitochondrial signaling

Since the cellular synthesis of glutathione is ATP-dependent, decreased ETC activity also undermines glutathione synthesis, further exacerbating oxidative stress in a vicious cycle [8]