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Initial case reports and small observational studies suggested a possible signal

GLP-1 drugs also called GLP-1 agonists were originally developed to treat type 2 diabetes

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Since mitochondrial oxidative phosphorylation depends on local NADH availability, whereas cytosolic NADPH supports reductive buffering, disproportionate redistribution between compartments may amplify localized redox imbalance even when total cellular NAD levels appear preserved