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GLP-1 receptors are expressed on up to 70% cerebral arterioles [18] and endothelial cells, and experimental models demonstrate that GLP-1 signaling promotes arteriolar vasodilation, improved endothelial nitric oxide signaling, and angiogenic pathways such as VEGF upregulation, all of which enhance nutrient and oxygen delivery to metabolically active brain regions [13,14]

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HF, a complex clinical syndrome characterized by significant functional impairment and high morbidity, stems from impaired cardiac contractility and/or ventricular filling [199]

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