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Proposed Mechanism BPC-157 appears to work through multiple pathways: Maintaining vesicular integrity (affecting VMATs and dopamine transporters) Possibly occupying its own vesicular storage sites Competing for dopamine receptor binding Functionally substituting when dopaminergic neurons are lost This last property, essentially filling in for missing dopamine function, would explain why the peptide counteracts both receptor agonists (apomorphine) and antagonists (haloperidol, fluphenazine, clozapine, sulpiride) with equal effectiveness
Growth Hormone Receptor Upregulation in Tendon Tissue Research shows BPC-157 dose- and time-dependently increases growth hormone receptor expression in tendon fibroblasts at both the mRNA and protein level Chang et al
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Progress in Neurobiology, 203, 102082
The next day, cells were washed with PBS and incubated with goat anti-mouse IgG AlexaFluor 488 (2 g/mL, A11029, Invitrogen) antibody diluted 1:1000 in PBS for 2 h at RT
