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These medications work by mimicking the action of naturally occurring incretin hormones, which stimulate insulin secretion in response to food intake, suppress glucagon release, slow gastric emptying, and promote satiety through central nervous system pathways

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In glutamate-treated HT22 cells, both NE and DA significantly suppressed glutathione depletion-associated mitochondrial dysfunction including mitochondrial superoxide accumulation, ATP depletion and mitochondrial AIF release