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Preclinical studies have shown that GLP-1 RAs can enhance endothelial nitric oxide (NO) production, promote vasodilation, and inhibit endothelial apoptosis and inflammation

We used cholesterol-enriched SH-SY5Y neuroblastoma cells, which we had previously characterized and shown the link between high cholesterol, reduced mitochondrial GSH levels, and greater susceptibility to the toxic A peptides [20, 51]

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In humans, a homozygous partial deletion of IGF-1R plays a neuroprotective role in various neurodegenerative disorders including Alzheimers and Huntington disease 27,28

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