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The degenerative environment of chronic tendinopathy lacks the active cellular repair response present in acute injuries, and BPC-157's ability to initiate fibroblast migration and growth factor receptor upregulation addresses the biological deficit underlying the chronic state

It works by inhibiting NNMT, leading to metabolic changes that can promote the use of fat for energy, reduce fat storage, and potentially slow down cellular aging

To detect any contamination by genomic DNA, the primers were designed to span intron-exon boundaries

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