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Utilizing proteomics, we identify elevated levels of Pin1 in preclinical AD (PCAD) brain [60], consistent with the notion that PCAD subjects, characterized by normal scores on tests of cognition but having AD-like pathology in brain, respond to elevated A by increasing expression of Pin1

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In erythroleukemia K562 cells, in addition to the above identified NRF2 binding ARE motif, there is also a strong association of active histone codes (such as H3K4Me3, H3K9Ac, H3K27Ac, H3K79Me2) upstream of the transcription start site (Supplementary Fig

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There are no head-to-head clinical trials proving one is superior to the other