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After amino acids rich meals, there is a higher increase in plasmatic GLP-1 than after meals rich in carbohydrates or lipids, with no difference between the last two

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ApoC-III is a key regulator of triglyceride metabolism, and human kinetic studies have shown that impaired catabolism of TRLs, linked to increased levels of plasma apoC-III, is the main determinant of plasma triglyceride levels in the population [8]