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Neuron-mediated regulation of CBF Neurons release chemical signals that directly or indirectly influence receptors on pericytes and astrocytes, resulting in either decreased CBF due to depolarization and cell contraction or increased CBF through hyperpolarization and cell relaxation

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Such activation triggers intracellular calcium transients, which in turn disrupt the localization and phosphorylation of both tight junction and adherens junction proteins [130, 131]

& Sun, H.-Z

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