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Therefore, as an alternative to URA3 (above), a strain (designated pGSH1-GAP1 in the following context) was constructed for using GAP1 as the counter-selectable marker to enrich phenotypically high-GSH producers, via replacement of the native pGAP1 promoter with pGSH1 (Fig

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Structural Deficiencies in Granuloma Formation in TNF Gene-Targeted Mice Underlie the Heightened Susceptibility to Aerosol Mycobacterium Tuberculosis Infection, Which Is Not Compensated for by Lymphotoxin