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Another potential mechanism suggested by experimental research is the potential GHK-Cu action on synthesis of matrix metalloproteinase-2 (MMP-2) and tissue inhibitors of metalloproteinases (TIMPs), which are deemed crucial for extracellular matrix remodeling

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(PubMed) There is also a more basic issue: even when these studies report health or survival benefits, they do not establish that the benefit was driven specifically by telomere extension