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Together, these compounds provide complementary pathways that may support enhanced outcomes in research models related to inflammation modulation, vascular support, and cellular turnover

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In this study, we explored new pathological mechanisms at the subcellular level that can potentially be used to address tendinopathy

The TA-CNPs increased cell proliferation lowered TGF- and COX-2 expression, and decreased MMP-1 expression (the primary enzyme for collagen degradation), all of which led to a reduction in the inflammatory reactions linked to UVB radiation

Next, we evaluated photocrosslinking between a photoreactive probe (PRP) (a slow inhibitor of TGR, Fig