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Although the free radical theory and the oxidative damage theory describe that accumulation of oxidative damage in cellular macromolecules is immensely toxic, ROS products by normal cell metabolism are vital to cell homeostasis maintenance, especially for its roles in immunocompetence and activation of several signal transduction pathways [18, 19]

In human metabolic research 3 , elevated IGF-1 levels correlated with improved muscle energy utilization and reduced fatigue during recovery phases

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These protective benefits have been observed in the GI tract, liver, cardiovascular system, and brain [20, 36]

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Under conditions of oxidative stress, this impact does not seem to result in increased cell numbers but instead offsets some of the detrimental impacts of ROS