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doi: 10.1001/jama.293.11.1338 75

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Proteins highly engaged by SCLA compounds were less labelled by subsequent treatment with an alkyne-modified probe, thereby showing a reduced signal following click chemistry, streptavidin pulldown and quantitative label-free MS (Fig

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317 Importantly, the methylation of YAP at K494, which is mediated by the SET1A methyltransferase complex, controls the activation of YAP by preventing its export from the nucleus to the cytoplasm