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Crucially, this study shifted the scientific paradigm from a simple model of "glutathione depletion" to a model of an "overwhelmed compensatory mechanism." The discovery that circulating immune cells in these patients actually have elevated glutathione levelswhile simultaneously suffering from a severe depletion of other protective enzymes like SOD2revealed that the cells are desperately, and unsuccessfully, trying to fight off a catastrophic level of oxidative damage

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J Neuroophthalmol 39(1):8293 De Simone R et al (2020) Headache in idiopathic intracranial hypertension A

Nickell et al., 2017)

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This is thought to be related to a transient vasodilation effect