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Biochemically, ferroptosis is indicated by the inhibition of cystine/glutamate antiporter (System Xc ), the depletion of glutathione (GSH), decreased activity of glutathione peroxidase 4 (GPX4), accumulation of lipid peroxides (LOOH), and a decline in mitochondrial membrane potential (Mao et al., 2021

This disease is caused by plaque accumulation within the arteries

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Both receptors display a differential expression in normal tissues

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Experimental models of bone defects have shown that, when SVVYGLR was implanted together with a collagen sponge, an upregulation of osteogenesis and angiogenesis was observed [169, 239]