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This deficiency specifically impairs mitochondrial Fe-S cluster proteins, leading to disruptions in electron transport chain activity, cellular energy metabolism, and cell cycle progression, ultimately resulting in growth inhibition and apoptosis

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TB500:~4963 g/mol

doi: 10.1007/s00204-019-02538-y

In humans, almost 4070% of clinically used drugs are subjected to glucuronidation (Wells et al., 2004)

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