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Accordingly, immunohistochemistry revealed that recombinant CXCL10 treatment sharply increased the level of p-FOXO3A in the nucleus of PMFs from the CXCL10 group, whereas IL18R1 antibody treatment reversed this increase to levels close to those of the control in the 10+18ab group (Fig
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After binding to its receptor (IL-22RA1), which is selectively expressed by non-hematopoietic cells (140), IL-22 induces phosphorylation of STAT-3
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