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Fluid shear stress induces EMT of circulating Tumor cells via JNK Signaling in Favor of their survival during Hematogenous Dissemination

Following the finding that the cancer cell lines did not elevate telomerase enzyme activity upon epitalon treatment but did elongate telomeres, we investigated the other commonly used mechanism to extend telomeres in mammals, ALT (Alternative Lengthening of Telomeres)

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