[DOI] [PMC free article] [PubMed] [Google Scholar] 12.McLean BA, Wong CK, Campbell JE, Hodson DJ, Trapp S, Drucker DJ
GLN1 is predicted to catalyse the amination of glutamate to form glutamine
307,308 Among the known pathogenic genes associated with HCM, mutations in MYBPC3 (myosin-binding protein C3) are the most prevalent, 309 with the majority being loss-of-function variants that result in haploinsufficiency
Unlike native IGF-1, the LR3 variant does not bind strongly to IGF Binding Proteins (IGFBPs)
Eur J Clin Microbiol Infect Dis 33:16531659 Van Hoof E, Cluydts R, De Meirleir K (2003) Atypical depression as a secondary symptom in chronic fatigue syndrome
Hiller N, Fritz-Wolf K, Deponte M, Wende W, Zimmermann H, et al