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This effect is probably a consequence of the increased baseline expression of caspase-1 and perhaps other inflammasome-associated factors, such as Gasdermins C2 and C3 that can act as terminal effectors of inflammatory cell lysis 27 , which were observed here in vitro and were previously described in ex vivo proteomics studies of mouse urothelia 8
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Biomarker Effects Were Present Despite clinical failure, semaglutide showed biological activity: CSF biomarkers including pTau181, pTau217, and others demonstrated nominally significant reductions of approximately 10% in treated patients
Although the effect was not as pronounced as in a preventive model in Figure 4, sulfasalazine treatment from day 30 partially reversed the formation of TLS (Supplemental Figure 8, BD), with a corresponding decrease in the expression of TLS-related molecules (Supplemental Figure 8E)
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