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It is probable that the long-term repercussions of neonatal hyperbilirubinemia could include potential genetic or carcinogenic ramifications later in life because DNA strand breaks have the potential to result in carcinogenic and mutagenic outcomes

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Finally, inhibition of GGT activity deprives tumours of cysteine and slows their growth

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The foundation of any successful claim requires establishing a clear causal relationship between the medication and the ocular injury through comprehensive medical documentation