In addition, mGluR5 inhibition rescued autistic phenotypes in mouse models of ASD
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Nanoparticles provide targeted therapeutic and diagnostic effects due to higher drug loading and bioavailability, lower dosing frequency, good biocompatibility and biodegradability, greater stability, fewer side effects, and less invasiveness[5, 6]
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Recently, specific inhibitors of ACSL4 and research on its upstream transcriptional regulators such as SP1 have shown clear cartilage-protective effects in OA models, suggesting that future strategies combining GPX4 modulation to enhance defense with ACSL4 inhibition to reduce attack may achieve more potent and synergistic therapeutic outcomes (He W
