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We believe these results are due to the nanomolar agonist concentrations applied, which were selected to simulate receptor activation levels similar to those that might occur physiologically, even if not precisely matching actual in vivo concentrations

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This indirectly suggests either a protective, or neutral, involvement of the oxidised forms of gliotoxin or sporidesmin in protecting against the deleterious effects of oxygen by selective protein inactivation

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CI, 0.72 0.91), both AD (HR, 0.78