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466 One study proposed a novel mechanism wherein postprandial GLP-1 release, under normal physiological conditions, inhibits the activity of chemosensory cells in the carotid body, thereby counteracting sympathetic excitability mediated by elevated blood glucose or insulin levels

While there was a substantial increase over the ten-year period between 2012 and 2022, some evidence indicates that utilization has only continued to rise over the last two years, beyond the most recent claims data available

AK was responsible for data acquisition and statistical analysis

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doi: 10.2337/dbi21-0004 64 FriasJPDaviesMJRosenstockJPerez ManghiFCFernandez LandoLBergmanBKet al

However, endogenous GLP-1 can be inactivated by dipeptidyl peptidase-4 (DPP-4) [4]