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Longer exposures of 2-month-old rats or 3-month-old APP/PS1 transgenic mice to sidestream cigarette smoke (1h/day x 5 days/wk x two or 4 months) induces tau and amyloid pathology like that reported in MCI and patients with dementia ( i.e ., insulin signaling, oxidative stress) and the accumulation of neuropathological proteins ( i.e ., tau, amyloid)

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This is a big percentage difference however there are also other factors that can be considered that may affect the drop-in success rate

The more someone smokes, and the higher the frequency of smoking, the longer nicotine takes to leave the body

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