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Mutant p53 proteins generally show intense phosphorylation and acetylation at sites that are well known to stabilize wild-type p53, and so could facilitate accumulation of dysfunctional mutant p53 in the nucleus, where it can act as an oncogene

10.1016/j.jacc.2016.03.569 [DOI] [PubMed] [Google Scholar] 16.Antoniewicz L, Kabele M, Nilsson U, Pourazar J, Rankin G, Bosson JA, et al

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